{"id":6812,"date":"2024-10-06T21:33:37","date_gmt":"2024-10-06T19:33:37","guid":{"rendered":"https:\/\/veganer-ernaehrungsberater.info\/?p=6812"},"modified":"2025-04-06T11:46:29","modified_gmt":"2025-04-06T09:46:29","slug":"cholesterin-und-fruktose","status":"publish","type":"post","link":"https:\/\/veganer-ernaehrungsberater.info\/en\/cholesterol-and-fructose\/","title":{"rendered":"Cholesterol and fructose"},"content":{"rendered":"\n
\n \t\t\t\t\n\t\t
\n\t\t\t\t\t
\n\t\t\t\t\t
\n\t\t\t\t\t\t\t\t\t
\n\t\t\t\t\n
\n\t

\n \n\t\t\t\t\tCholesterol and fructose\t\t <\/span>\n\n\t\n \n\t\t\t\t\tThe Hidden Danger of Fructose\t\t <\/span>\n <\/h1>\n<\/div>\n\n\n
\n
\n

How VLDL and sdLDL Increase the Risk of Cardiovascular Disease<\/h2>

Fat Formation and the Role of VLDL, LDL, and sdLDL<\/h3>

Excessive fructose consumption can trigger an unhealthy cycle in our bodies, particularly through the conversion of fructose into triglycerides<\/strong> \u2014a type of fat that is stored in our cells. Once fructose reaches the liver, the metabolic process begins, during which a large portion of the fructose is converted into fat. This fat is either stored directly in the liver, leading to non-alcoholic fatty liver disease<\/strong> , or it is released into the bloodstream in the form of VLDL<\/strong> (very-low-density lipoprotein) particles.<\/p> <\/div>\n<\/div>\n\n

\n
\n\t\t \"What\t\n\t\t<\/div>\n\t\n \n \t<\/div>\n\t\t\t\t<\/div>\n\t\t\t\t\t\t<\/div>\n\t\t\t\t\t\t<\/div>\n\t\t<\/div>\n\t\t\t\t\t\t\t\t\n\t\t
\n\t\t\t\t\t
\n\t\t\t\t\t
\n\t\t\t\t\t\t\t\t\t
\n\t\t\t\t\n
\n
\n

VLDL: Fat-Carrying Buoys<\/h3>

VLDL particles are fat-laden transport vehicles responsible for carrying triglycerides from the liver to other parts of the body, where they are either used for energy or stored in fat cells. You can think of these VLDL particles as \u201cfat-carrying buoys<\/strong>\u201d that float through the bloodstream, dropping off their cargo and gradually becoming smaller. However, when the liver becomes heavily fatty\u2014often due to high fructose consumption\u2014it produces especially large VLDL particles that carry a lot of fat. These \"especially large buoys\" shrink after delivering their fat load into extremely small and dense sdLDL<\/strong> (small dense LDL) particles, which are known as particularly dangerous \"bad cholesterol.<\/strong>\" These small, dense LDL particles can more easily lodge in artery walls and cause inflammation.<\/p> <\/div>\n<\/div>\n\t\t\t\t<\/div>\n\t\t\t\t\t\t<\/div>\n\t\t\t\t\t\t<\/div>\n\t\t<\/div>\n\t\t\t\t\t\t\t\t\n\t\t

\n\t\t\t\t\t
\n\t\t\t\t\t
\n\t\t\t\t\t\t\t\t\t
\n\t\t\t\t\t\t
\n\t\t\t\t\t
\n\t\t\t\t\t
\n\t\t\t\t\t\t\t\t\t
\n\t\t\t\t\n
\n
\n\t\t \t\n\t\t<\/div>\n\t\n \n \t<\/div>\n\t\t\t\t<\/div>\n\t\t\t\t\t\t<\/div>\n\t\t\t\t\t
\n\t\t\t\t\t\t\t\t\t
\n\t\t\t\t\n
\n
\n

Fructose: The Hidden Sugar<\/h3>\n

Fructose is found in many processed foods, often in the form of corn syrup or added sugar, and is one of the main sources of hidden sugars in our diet.<\/p> <\/div>\n<\/div>\n\t\t\t\t<\/div>\n\t\t\t\t\t\t<\/div>\n\t\t\t\t\t

\n\t\t\t\t\t\t\t\t\t
\n\t\t\t\t\n
\n
\n\t\t \"Visceral\t\n\t\t<\/div>\n\t\n \n \t<\/div>\n\t\t\t\t<\/div>\n\t\t\t\t\t\t<\/div>\n\t\t\t\t\t
\n\t\t\t\t\t\t\t\t\t
\n\t\t\t\t\n
\n
\n

Visceral Fat: The Invisible Danger<\/strong><\/h3>\n

The fat produced by fructose is often stored as visceral fat, which surrounds the organs and increases the risk of metabolic diseases.<\/p>\n

\u00a0<\/p> <\/div>\n<\/div>\n\t\t\t\t<\/div>\n\t\t\t\t\t\t<\/div>\n\t\t\t\t\t\t<\/div>\n\t\t<\/div>\n\t\t\t\t\t\t

\n\t\t\t\t\t
\n\t\t\t\t\t
\n\t\t\t\t\t\t\t\t\t
\n\t\t\t\t\n
\n
\n

The liver and fructose<\/strong><\/h3>\n

Fructose is converted into fat in the liver. High fructose consumption leads to fatty liver and increases the risk of insulin resistance.<\/p> <\/div>\n<\/div>\n\t\t\t\t<\/div>\n\t\t\t\t\t\t<\/div>\n\t\t\t\t\t

\n\t\t\t\t\t\t\t\t\t
\n\t\t\t\t\n
\n
\n\t\t \"The\t\n\t\t<\/div>\n\t\n \n \t<\/div>\n\t\t\t\t<\/div>\n\t\t\t\t\t\t<\/div>\n\t\t\t\t\t
\n\t\t\t\t\t\t\t\t\t
\n\t\t\t\t\n
\n
\n

Fructose and cholesterol<\/strong><\/h3>\n

The breakdown of fructose creates VLDL particles<\/strong>, which become LDL (bad cholesterol)<\/strong> in the body. This increases the risk of arteriosclerosis and heart disease.<\/p> <\/div>\n<\/div>\n\t\t\t\t<\/div>\n\t\t\t\t\t\t<\/div>\n\t\t\t\t\t

\n\t\t\t\t\t\t\t\t\t
\n\t\t\t\t\n
\n
\n\t\t \"Fructose\t\n\t\t<\/div>\n\t\n \n \t<\/div>\n\t\t\t\t<\/div>\n\t\t\t\t\t\t<\/div>\n\t\t\t\t\t\t<\/div>\n\t\t<\/div>\n\t\t\t\t\t\t\t\t<\/div>\n\t\t\t\t\t\t<\/div>\n\t\t\t\t\t\t<\/div>\n\t\t<\/div>\n\t\t\t\t\t\t\t\t\n\t\t
\n\t\t\t\t\t
\n\t\t\t\t\t
\n\t\t\t\t\t\t\t\t\t
\n\t\t\t\t\n
\n
\n

LDL and sdLDL: The dangerous cholesterol particles<\/h3>\n

LDL particles, especially the so-called sdLDL (small dense LDL), pose a particular danger. These small, dense LDL particles are much more harmful than normal LDL particles because they have a greater tendency to penetrate artery walls. There, they cause inflammation<\/strong> and promote the formation of plaque\u2014deposits that narrow the arteries and dramatically increase the risk of Cardiovascular diseases<\/strong>, such as heart attacks and strokes.\n\nThe buoy analogy illustrates this process: Imagine VLDL particles as large, fat-laden buoys that are loaded with fat molecules. As they unload their cargo, they shrink into LDL particles that continue their journey through the bloodstream. sdLDL particles<\/strong>, which are small and dense, resemble \"fine sand\" that settles into the smallest crevices of the artery walls. They can penetrate deep into the endothelium<\/strong> (the innermost layer of blood vessels), causing inflammation that plays a critical role in the development of atherosclerosis<\/strong> What makes this particularly problematic is that, due to fat overload in the liver, these VLDL particles become extremely large. After delivering their fat load, they shrink into particularly small, dense sdLDL particles, which are especially dangerous. These small particles are the ones that lodge in the artery walls, promoting plaque formation, which narrows the arteries and significantly increases the risk of cardiovascular diseases.<\/p> <\/div>\n<\/div>\n\t\t\t\t<\/div>\n\t\t\t\t\t\t<\/div>\n\t\t\t\t\t\t<\/div>\n\t\t<\/div>\n\t\t\t\t\t\t\t\t\n\t\t

\n\t\t\t\t\t
\n\t\t\t\t\t
\n\t\t\t\t\t\t\t\t\t
\n\t\t\t\t\t\t
\n\t\t\t\t\t
\n\t\t\t\t\t
\n\t\t\t\t\t\t\t\t\t
\n\t\t\t\t\n
\n
\n\t\t \"LDL:\t\n\t\t<\/div>\n\t\n \n \t<\/div>\n\t\t\t\t<\/div>\n\t\t\t\t\t\t<\/div>\n\t\t\t\t\t
\n\t\t\t\t\t\t\t\t\t
\n\t\t\t\t\n
\n
\n

LDL: The bad cholesterol<\/strong><\/h3>\n

LDL<\/strong> builds up in the arteries and increases the risk of cardiovascular disease. High LDL levels are often caused by too much fructose and saturated fatty acids.<\/p> <\/div>\n<\/div>\n\t\t\t\t<\/div>\n\t\t\t\t\t\t<\/div>\n\t\t\t\t\t

\n\t\t\t\t\t\t\t\t\t
\n\t\t\t\t\n
\n
\n\t\t \"HDL:\t\n\t\t<\/div>\n\t\n \n \t<\/div>\n\t\t\t\t<\/div>\n\t\t\t\t\t\t<\/div>\n\t\t\t\t\t
\n\t\t\t\t\t\t\t\t\t
\n\t\t\t\t\n
\n
\n

HDL: The good cholesterol<\/strong><\/h3>\n

HDL<\/strong> helps transport excess cholesterol back to the liver where it is broken down. A healthy HDL level can reduce the risk of cardiovascular disease.<\/p> <\/div>\n<\/div>\n\t\t\t\t<\/div>\n\t\t\t\t\t\t<\/div>\n\t\t\t\t\t\t<\/div>\n\t\t<\/div>\n\t\t\t\t\t\t

\n\t\t\t\t\t
\n\t\t\t\t\t
\n\t\t\t\t\t\t\t\t\t
\n\t\t\t\t\n
\n
\n

sdLDL: The Real Enemy<\/strong><\/h3>\n

Small, dense sdLDL<\/strong>particles are particularly dangerous because they easily penetrate the artery walls and cause inflammation, leading to plaque formation.<\/p> <\/div>\n<\/div>\n\t\t\t\t<\/div>\n\t\t\t\t\t\t<\/div>\n\t\t\t\t\t

\n\t\t\t\t\t\t\t\t\t
\n\t\t\t\t\n
\n
\n\t\t \"sdLDL:\t\n\t\t<\/div>\n\t\n \n \t<\/div>\n\t\t\t\t<\/div>\n\t\t\t\t\t\t<\/div>\n\t\t\t\t\t
\n\t\t\t\t\t\t\t\t\t
\n\t\t\t\t\n
\n
\n

The balance between LDL and HDL<\/strong><\/h3>\n

A healthy ratio of HDL to LDL<\/strong> is crucial. High fructose consumption can disrupt this balance and weaken HDL effectiveness.<\/p> <\/div>\n<\/div>\n\t\t\t\t<\/div>\n\t\t\t\t\t\t<\/div>\n\t\t\t\t\t

\n\t\t\t\t\t\t\t\t\t
\n\t\t\t\t\n
\n
\n\t\t \"The\t\n\t\t<\/div>\n\t\n \n \t<\/div>\n\t\t\t\t<\/div>\n\t\t\t\t\t\t<\/div>\n\t\t\t\t\t\t<\/div>\n\t\t<\/div>\n\t\t\t\t\t\t\t\t<\/div>\n\t\t\t\t\t\t<\/div>\n\t\t\t\t\t\t<\/div>\n\t\t<\/div>\n\t\t\t\t\t\t\t\t\n\t\t
\n\t\t\t\t\t
\n\t\t\t\t\t
\n\t\t\t\t\t\t\t\t\t
\n\t\t\t\t\n
\n
\n

How fructose is metabolized in the liver<\/h2>

fructose<\/strong> is Processed Differently\nWhile glucose can be directly used by nearly all body cells, fructose must first be metabolized in the liver. This is because the body lacks specific transporters for fructose to enter cells directly, as is the case with glucose.<\/p>

When we consume fructose\u2014whether from fruit or added sugar in processed foods\u2014it is transported to the liver, where it can follow two primary pathways:<\/p>